Ersin Kasim Ulusoy
Rhabdomyolysis is a condition in which intracellular components undergo systemic circulation leading to impairment in clinical and laboratory findings due to striated muscle destruction due to traumatic or traumatic causes. The most common clinical symptoms of rhabdomyolysis are muscle weakness, muscle pain and dark urinary tract. The most important complication is acute renal failure.
A 32-year-old female patient was brought in by an immediate family member with complaints of fatigue, muscle aches, and wandering in the morning. The patient had a history of psychosis and the use of antipsychotic medication. In the patient’s anamnesis, it was learned that she jumped about six meters in height 10 days ago for suicide but was able to walk completely independently and without support. There was flask paralysis in the lower extremity on the neurological examination performed. She was begun to be followed by Guillain barre syndrome (GBS] by preliminary diagnosis. In the biochemical analysis results, creatine kinase 50,902 U/L (24-190), AST 871 U/L (10-40) and lactate dehydrogenase 1617 U/L (220-450) were detected. According to the clinical and laboratory results, acute renal insufficiency (ARI] developed in the patient despite diagnosis of rhabdomyolysis and intensive hydration therapy. ARI was completely improved with appropriate treatment. Patient was mobilized and discharged.
In this article, a case is presented in which GBS was considered with the initial examination findings in the emergency department and clinical and laboratory findings and rhabdomyolysis were detected in follow-up. It was emphasized the importance of seeing rhabdomyolysis, which is among the neuromusculer aciller, in mind for the rare occurrence. The presence of flask paralysis in our case clinic and the development of rhabdomyolysis after falling from the top have the feature of being first.
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